Ca2+ sensitizer superior to catecholamine during myocardial stunning?☆

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Ca2+ sensitizer superior to catecholamine during myocardial stunning?

BACKGROUND After open-chest cardiac surgery, ventricular function remains depressed (myocardial stunning). Catecholamines (epinephrine) improve ventricular function by increasing the intracellular Ca(2+) concentration. In parallel, the oxygen consumption is increased, so that the hitherto intact myocardium can be jeopardized. In the very insufficient ventricle, epinephrine can even become ineff...

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Myocardial Stunning in Dogs

This study was undertaken to elucidate whether cal-adrenoceptor activity is beneficial to contractile dysfunction during reperfusion after a brief period of ischemia (stunned myocardium) in 54 open-chest dogs. Contractile dysfunction assessed by fractional shortening (FS) was observed 3 hours after the onset of reperfusion following 15 minutes of complete occlusion of the left anterior descendi...

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Mechanism of myocardial "stunning".

Among the numerous mechanisms proposed for myocardial stunning, three appear to be more plausible: 1) generation of oxygen radicals, 2) calcium overload, and 3) excitation-contraction uncoupling. First, the evidence for a pathogenetic role of oxygen-derived free radicals in myocardial stunning is overwhelming. In the setting of a single 15-minute coronary occlusion, mitigation of stunning by an...

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ژورنال

عنوان ژورنال: European Journal of Cardio-Thoracic Surgery

سال: 2008

ISSN: 1010-7940

DOI: 10.1016/j.ejcts.2008.04.042